selected publications
-
Inborn errors of OAS-RNase L in SARS-CoV-2-related multisystem inflammatory syndrome in children.
Science (New York, N.Y.).
2023
Academic Article
GET IT
Times cited: 13 - hiPSCs as a Unique Platform to Model Cardiogenesis in NOTCH1-Associated HLHS: hiPSCs to Model Complex Congenital Heart Defects. Circulation research. 2023 Editorial Article GET IT
-
Cardiomyocytes recruit monocytes upon SARS-CoV-2 infection by secreting CCL2.
Stem cell reports.
2021
Article
GET IT
Times cited: 3 -
Constitutively Activating GNAS Somatic Mutation in Right Ventricular Outflow Tract Tachycardia.
2021
GET IT
Times cited: 2 -
Cardiomyocytes recruit monocytes upon SARS-CoV-2 infection by secreting CCL2.
Stem cell reports.
2021
Academic Article
GET IT
Times cited: 26 -
An Immuno-Cardiac Model for Macrophage-Mediated Inflammation in COVID-19 Hearts.
Circulation research.
2021
Academic Article
GET IT
Times cited: 25 - SARS-CoV-2 Infected Cardiomyocytes Recruit Monocytes by Secreting CCL2. Research square. 2020 Article GET IT
-
Modeling polymorphic ventricular tachycardia at rest using patient-specific induced pluripotent stem cell-derived cardiomyocytes.
EBioMedicine.
2020
Academic Article
GET IT
Times cited: 14 -
A Human Pluripotent Stem Cell-based Platform to Study SARS-CoV-2 Tropism and Model Virus Infection in Human Cells and Organoids.
Cell stem cell.
2020
Academic Article
GET IT
Times cited: 420 -
Inducible Pluripotent Stem Cell-Derived Cardiomyocytes Reveal Aberrant Extracellular Regulated Kinase 5 and Mitogen-Activated Protein Kinase Kinase 1/2 Signaling Concomitantly Promote Hypertrophic Cardiomyopathy in RAF1-Associated Noonan Syndrome.
Circulation.
2019
Academic Article
GET IT
Times cited: 38 -
Generation of an induced pluripotent stem cell line (TRNDi003-A) from a Noonan syndrome with multiple lentigines (NSML) patient carrying a p.Q510P mutation in the PTPN11 gene.
Stem cell research.
2018
Academic Article
GET IT
Times cited: 9 -
RhoA signaling in cardiomyocytes protects against stress-induced heart failure but facilitates cardiac fibrosis.
Science signaling.
2014
Academic Article
GET IT
Times cited: 59 -
The role of the protein tyrosine phosphatase SHP2 in cardiac development and disease.
Seminars in cell & developmental biology.
2014
Review
GET IT
Times cited: 32 -
β-adrenergic receptor stimulation transactivates protease-activated receptor 1 via matrix metalloproteinase 13 in cardiac cells.
Circulation.
2012
Academic Article
GET IT
Times cited: 71 -
Stimulating healthy tissue regeneration by targeting the 5-HT₂B receptor in chronic liver disease.
Nature medicine.
2011
Academic Article
GET IT
Times cited: 166 -
Cyclophilin A promotes cardiac hypertrophy in apolipoprotein E-deficient mice.
Arteriosclerosis, thrombosis, and vascular biology.
2011
Academic Article
GET IT
Times cited: 67 -
Serotonin and angiotensin receptors in cardiac fibroblasts coregulate adrenergic-dependent cardiac hypertrophy.
Circulation research.
2008
Academic Article
GET IT
Times cited: 99 -
Serotonin 5-HT(2B) receptor blockade prevents reactive oxygen species-induced cardiac hypertrophy in mice.
Hypertension (Dallas, Tex. : 1979).
2008
Academic Article
GET IT
Times cited: 41 -
Involvement of the serotonin 5-HT2B receptor in cardiac hypertrophy linked to sympathetic stimulation: control of interleukin-6, interleukin-1beta, and tumor necrosis factor-alpha cytokine production by ventricular fibroblasts.
Circulation.
2004
Academic Article
GET IT
Times cited: 107 -
[The 5-HT2B receptor: a main cardio-pulmonary target of serotonin].
Journal de la Societe de biologie.
2004
Review
GET IT
Times cited: 10 -
Overexpression of the serotonin 5-HT2B receptor in heart leads to abnormal mitochondrial function and cardiac hypertrophy.
Circulation.
2003
Academic Article
GET IT
Times cited: 117