Altamirano, Francisco

Publications

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selected publications

Polycystin-1 is required for insulin-like growth factor 1-induced cardiomyocyte hypertrophy. PloS one. 2021 Academic Article GET IT
Times cited: 3
Polycystin-1 regulates cardiomyocyte mitophagy. FASEB journal : official publication of the Federation of American Societies for Experimental Biology. 2021 Academic Article GET IT
Times cited: 6
Activation of Autophagic Flux Blunts Cardiac Ischemia/Reperfusion Injury. Circulation research. 2021 Academic Article GET IT
Times cited: 20
NAD⁺ Repletion Reverses Heart Failure With Preserved Ejection Fraction. Circulation research. 2021 Academic Article GET IT
Times cited: 51
Xbp1s-FoxO1 axis governs lipid accumulation and contractile performance in heart failure with preserved ejection fraction. Nature communications. 2021 Academic Article GET IT
Times cited: 38
Epigenetic Reader BRD4 (Bromodomain-Containing Protein 4) Governs Nucleus-Encoded Mitochondrial Transcriptome to Regulate Cardiac Function. Circulation. 2020 Academic Article GET IT
Times cited: 29
PKD2/polycystin-2 induces autophagy by forming a complex with BECN1. Autophagy. 2020 Academic Article GET IT
Times cited: 15
FoxO1-Dio2 signaling axis governs cardiomyocyte thyroid hormone metabolism and hypertrophic growth. Nature communications. 2020 Academic Article GET IT
Times cited: 23
Remodeling of substrate consumption in the murine sTAC model of heart failure. Journal of molecular and cellular cardiology. 2019 Academic Article GET IT
Times cited: 13
Polycystin-1 Assembles With Kv Channels to Govern Cardiomyocyte Repolarization and Contractility. Circulation. 2019 Academic Article GET IT
Times cited: 20
Fibroblast Primary Cilia Are Required for Cardiac Fibrosis. Circulation. 2019 Academic Article GET IT
Times cited: 75
Nitrosative stress drives heart failure with preserved ejection fraction. Nature. 2019 Academic Article GET IT
Times cited: 345
Is malignant hyperthermia associated with hyperglycaemia?. British journal of anaesthesia. 2018 Letter GET IT
Times cited: 8
Polycystin-2-dependent control of cardiomyocyte autophagy. Journal of molecular and cellular cardiology. 2018 Academic Article GET IT
Times cited: 22
Down Syndrome Critical Region 1 Gene, Rcan1, Helps Maintain a More Fused Mitochondrial Network. Circulation research. 2018 Academic Article GET IT
Times cited: 45
Dysregulation of Intracellular Ca²⁺ in Dystrophic Cortical and Hippocampal Neurons. Molecular neurobiology. 2016 Academic Article GET IT
Times cited: 17
Whole Body Periodic Acceleration Improves Muscle Recovery after Eccentric Exercise. Medicine and science in sports and exercise. 2016 Academic Article GET IT
Times cited: 11
NOX2 Inhibition Impairs Early Muscle Gene Expression Induced by a Single Exercise Bout. Frontiers in physiology. 2016 Academic Article GET IT
Times cited: 50
Chronic heart failure: Ca(2+), catabolism, and catastrophic cell death. Biochimica et biophysica acta. 2016 Review GET IT
Times cited: 20
Cardioprotection in ischaemia-reperfusion injury: novel mechanisms and clinical translation. The Journal of physiology. 2015 Review GET IT
Times cited: 35
Antioxidant Properties of Whole Body Periodic Acceleration (pGz). PloS one. 2015 Academic Article GET IT
Times cited: 17
Altered ROS production, NF-κB activation and interleukin-6 gene expression induced by electrical stimulation in dystrophic mdx skeletal muscle cells. Biochimica et biophysica acta. 2015 Academic Article GET IT
Times cited: 49
Non-invasive technology that improves cardiac function after experimental myocardial infarction: Whole Body Periodic Acceleration (pGz). PloS one. 2015 Academic Article GET IT
Times cited: 9
Age-dependent changes in diastolic Ca(2+) and Na(+) concentrations in dystrophic cardiomyopathy: Role of Ca(2+) entry and IP3. Biochemical and biophysical research communications. 2014 Academic Article GET IT
Times cited: 32
Whole body periodic acceleration is an effective therapy to ameliorate muscular dystrophy in mdx mice. PloS one. 2014 Academic Article GET IT
Times cited: 19
Ca2+ influx via the Na+/Ca2+ exchanger is enhanced in malignant hyperthermia skeletal muscle. The Journal of biological chemistry. 2014 Academic Article GET IT
Times cited: 21
Na+ dysregulation coupled with Ca2+ entry through NCX1 promotes muscular dystrophy in mice. Molecular and cellular biology. 2014 Academic Article GET IT
Times cited: 28
Nifedipine treatment reduces resting calcium concentration, oxidative and apoptotic gene expression, and improves muscle function in dystrophic mdx mice. PloS one. 2013 Academic Article GET IT
Times cited: 40
Testosterone signals through mTOR and androgen receptor to induce muscle hypertrophy. Medicine and science in sports and exercise. 2013 Academic Article GET IT
Times cited: 100
Cav1.1 controls frequency-dependent events regulating adult skeletal muscle plasticity. Journal of cell science. 2013 Academic Article GET IT
Times cited: 46
Electrical stimuli release ATP to increase GLUT4 translocation and glucose uptake via PI3Kγ-Akt-AS160 in skeletal muscle cells. Diabetes. 2012 Academic Article GET IT
Times cited: 90
Increased resting intracellular calcium modulates NF-κB-dependent inducible nitric-oxide synthase gene expression in dystrophic mdx skeletal myotubes. The Journal of biological chemistry. 2012 Academic Article GET IT
Times cited: 69
Malignant hyperthermia susceptibility arising from altered resting coupling between the skeletal muscle L-type Ca2+ channel and the type 1 ryanodine receptor. Proceedings of the National Academy of Sciences of the United States of America. 2012 Academic Article GET IT
Times cited: 71
Measurement of calcium release due to inositol trisphosphate receptors in skeletal muscle. Methods in molecular biology (Clifton, N.J.). 2012 Academic Article GET IT
Times cited: 7
Testosterone induces cardiomyocyte hypertrophy through mammalian target of rapamycin complex 1 pathway. The Journal of endocrinology. 2009 Academic Article GET IT
Times cited: 83
RpoS and RpoN are involved in the growth-dependent regulation of rfaH transcription and O antigen expression in Salmonella enterica serovar Typhi. Microbial pathogenesis. 2004 Academic Article GET IT
Times cited: 24

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Background

education and training

Ph.D., University of Chile (Chile) 2012
B.S., University of Chile (Chile) 2007

Contact

full name

Francisco Altamirano

primary email

fra4004@med.cornell.edu

VIVO Weill Cornell Medical College

Francisco Altamirano Assistant Professor of Molecular Biology in Cardiothoracic Surgery

Publications

selected publications

Background

education and training

Contact

full name

primary email